With such medications, besides behavioural changes, how are they able to measure outcomes without fMRIs? Like knowing whether neuron connections are amplified or not?
They don't, this is speculative (i.e. a theory) and almost certainly untrue (or a gross over-simplification), much like the early and now disproven serotonin theories of depression.
They don't, this is speculative (i.e. a theory) and almost certainly untrue (or a gross over-simplification), much like the early and now disproven serotonin theories of depression.